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Metastasis-associated MCL1 and P16 copy number alterations dictate resistance to vemurafenib in a BRAFV600E patient-derived papillary thyroid carcinoma preclinical model
Oncotarget (2015)
  • Mark Duquette, Harvard University
  • Peter M. Sadow, Harvard University
  • Amjad Husain, Harvard University
  • Jennifer N. Sims, Harvard University
  • Zeus A. Antonello, Harvard University
  • Andrew H. Fischer, University of Massachusetts Medical School
  • Chen Song, Harvard University
  • Elena Castellanos-Rizaldos, Harvard University
  • G. Mike Makrigiorgos, Harvard University
  • Junichi Kurebayashi, Kawasaki Medical School
  • Vania Nose, Harvard University
  • Paul Van Hummelen, Harvard University
  • Roderick T. Bronson, Harvard University
  • Michelle Vinco, University of Michigan
  • Thomas J. Giordano, University of Michigan
  • Dora Dias-Santagata, Harvard University
  • Pier Paolo Pandolfi, Harvard University
  • Carmelo Nucera, Harvard University
Publication Date
December 15, 2015
DOI
10.18632/oncotarget.6442
Citation Information
Mark Duquette, Peter M. Sadow, Amjad Husain, Jennifer N. Sims, et al.. "Metastasis-associated MCL1 and P16 copy number alterations dictate resistance to vemurafenib in a BRAFV600E patient-derived papillary thyroid carcinoma preclinical model" Oncotarget Vol. 6 Iss. 40 (2015) p. 42445 - 42467
Available at: http://works.bepress.com/zeus-antonello/5/