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Article
MYC and Twist1 cooperate to drive metastasis by eliciting crosstalk between cancer and innate immunity.
eLife (2020)
  • Renumathy Dhanasekaran, Stanford University
  • Virginie Baylot, Stanford University
  • Minsoon Kim, Stanford University
  • Sibu Kuruvilla, Stanford University
  • David I Bellovin, Stanford University
  • Nia Adeniji, Stanford University
  • Anand Rajan Kd, University of Iowa Hospitals and Clinics
  • Ian Lai, Stanford University
  • Meital Gabay, Stanford University
  • Ling Tong, Stanford University
  • Maya Krishnan, Stanford University
  • Jangho Park, Stanford University
  • Theodore Hu, Stanford University
  • Mustafa A Barbhuiya, Radiation Oncology and Molecular Radiation Sciences, Johns Hopkins University School of Medicine, Baltimore, United States.
  • Andrew J Gentles, Stanford University
  • Kasthuri Kannan, New York University
  • Phuoc T Tran, Radiation Oncology and Molecular Radiation Sciences, Johns Hopkins University School of Medicine, Baltimore, United States.
  • Dean W Felsher, Stanford University
Disciplines
Publication Date
January 14, 2020
DOI
10.7554/ELIFE.50731
Citation Information
Renumathy Dhanasekaran, Virginie Baylot, Minsoon Kim, Sibu Kuruvilla, et al.. "MYC and Twist1 cooperate to drive metastasis by eliciting crosstalk between cancer and innate immunity." eLife Vol. 9 (2020)
Available at: http://works.bepress.com/mustafa-barbhuiya/2/