"Y1767C, a novel SCN5A mutation, induces a persistent Na+ current and potentiates ranolazine inhibition of Nav1.5 channels" by Hai Huang

Article

Y1767C, a novel SCN5A mutation, induces a persistent Na+ current and potentiates ranolazine inhibition of Nav1.5 channels

American Journal of Physiology-heart and Circulatory Physiology (2011)

Hai Huang, Golden Village
Silvia G. Priori, University of Pavia
Carlo Napolitano, University of Pavia
Michael E. O'Leary, Thomas Jefferson University
Mohamed Chahine, Laval University

Link

Disciplines

Biology,
Pharmacology,
Anesthesiology and
Genetics

Publication Date

January 1, 2011

DOI

10.1152/ajpheart.00539.2010

Citation Information

Hai Huang, Silvia G. Priori, Carlo Napolitano, Michael E. O'Leary, et al.. "Y1767C, a novel SCN5A mutation, induces a persistent Na+ current and potentiates ranolazine inhibition of Nav1.5 channels" American Journal of Physiology-heart and Circulatory Physiology Vol. 300 Iss. 1 (2011)
Available at: http://works.bepress.com/michael-oleary/2/