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Triheptanoin for glucose transporter type I deficiency (G1D): modulation of human ictogenesis, cerebral metabolic rate, and cognitive indices by a food supplement.
JAMA Neurology (2014)
  • Juan M. Pascual, Rare Brain Disorders Program, Department of Neurology and Neurotherapeutics, The University of Texas Southwestern Medical Center, Dallas
  • Peiying Liu, University of Texas Southwestern Medical Center
  • Deng Mao, University of Texas Southwestern Medical Center
  • Dorothy I. Kelly, University of Texas Southwestern Medical Center
  • Ana Hernandez, Children's Medical Center of Dallas
  • Min Sheng, University of Texas Southwestern Medical Center
  • Levi B. Good, University of Texas Southwestern Medical Center
  • Qian Ma, University of Texas Southwestern Medical Center
  • Isaac Marin-Valencia, University of Texas Southwestern Medical Center
  • Xuchen Zhang, University of Texas Southwestern Medical Center
  • Jason Y. Park, University of Texas Southwestern Medical Center
  • Jason Y. Park, Children's Medical Center of Dallas
  • Linda S. Hynan, University of Texas at Austin
  • Peter Stavinoha, University of Texas Southwestern Medical Center
  • Peter Stavinoha, Children's Medical Center of Dallas
  • Charles R. Roe, University of Texas Southwestern Medical Center
  • Hanzhang Lu, University of Texas at Austin
Publication Date
October 1, 2014
DOI
10.1001/jamaneurol.2014.1584
Citation Information
Juan M. Pascual, Peiying Liu, Deng Mao, Dorothy I. Kelly, et al.. "Triheptanoin for glucose transporter type I deficiency (G1D): modulation of human ictogenesis, cerebral metabolic rate, and cognitive indices by a food supplement." JAMA Neurology Vol. 71 Iss. 10 (2014) p. 1255 - 1265
Available at: http://works.bepress.com/levi-good/5/