Dr. Troy Rohn has been on the faculty at Boise State University since 2000. He
graduated in 1990 from the University of California at Davis with a B.S. in Physiology,
and received his Ph.D. in Pharmacology from the University of Washington, Seattle, in
1994. His interests include the role of apoptosis in neurodegenerative diseases. Dr. Rohn
had several Postdoctoral stints including two plus years living in Paris, France, one
year at Montana State University in Bozeman, Montana, and two years at UC Irvine at the
Institute of Brain Aging and Dementia under the direction of Dr. Carl Cotman. Dr. Rohn
continues to collaborate extensively with UC Irvine and more specifically with Dr.
Elizabeth Head. He has obtained extramural funding continuously since his arrival at BSU
including grants from NIH, AFAR and most recently from AHAF. 

Articles

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The Triggering Receptor Expressed on Myeloid Cells 2: “TREM-ming” the Inflammatory Component Associated with Alzheimer's Disease, Oxidative Medicine and Cellular Longevity (2013)

Alzheimer's disease (AD) is an age-related neurodegenerative disorder characterized by a progressive loss of memory...

 

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Caspase-Cleaved Glial Fibrillary Acidic Protein Within Cerebellar White Matter of the Alzheimer's Disease Brain (with Lindsey W. Catlin and Wayne W. Poon), International Journal of Clinical and Experimental Pathology (2013)

Although the cerebellum is generally thought of as an area spared of Alzheimer's disease (AD)...

 

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Identification of an Amino-Terminal Fragment of Apolipoprotein E4 that Localizes to Neurofibrillary Tangles of the Alzheimer’s Disease Brain (with Lindsey W. Catlin, Kendra G. Coonse, and Jeffrey W. Habig), Brain Research (2012)

Although the risk factor for harboring the apolipoprotein E4 (apoE4) allele in late-onset Alzheimer’s disease...

 

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Targeting Alpha-Synuclein for the Treatment of Parkinson's Disease, CNS & Neurological Disorders - Drug Targets (2012)

Parkinson's disease (PD) is characterized as a neurodegenerative movement disorder presenting with rigidity, resting tremor,...

 

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Depletion of Beclin-1 Due to Proteolytic Cleavage by Caspases in the Alzheimer's Disease Brain (with Ellen Wirawan, Raquel J. Brown, Jordan R. Harris, Eliezer Masliah, and Peter Vandenabeele), Neurobiology of Disease (2011)

The Beclin-1 protein is essential for the initiation of autophagy and recent studies suggest this...

 

Presentations

Caspase-cleavage of the Amyloid Precursor Protein is Prevented After Overexpression of Bcl-2 in a Triple Transgenic Mouse Model of Alzheimer’s Disease (with D. K. Kumasada, V. Galvan, and E. Head), Eleventh Annual International Conference on Alzheimer’s Disease (2008)