"Occurrence of Mitochondria-targeted Late Embryogenesis Abundant (LEA) Gene in Animals Increases Organelle Resistance to Water Stress" by Michael A Menze

Article

Occurrence of Mitochondria-targeted Late Embryogenesis Abundant (LEA) Gene in Animals Increases Organelle Resistance to Water Stress

Journal of Biological Chemistry (2009)

Michael A Menze, University of Louisville
Leaf Boswell
Mehmet Toner, Harvard Medical School
Steven C. Hand

Link

Abstract

Anhydrobiotic animals survive virtually complete loss of cellular water. The mechanisms that explain this phenomenon are not fully understood but often include the accumulation of low molecular weight solutes such as trehalose and macromolecules like Late Embryogenesis Abundant (LEA) proteins. Here we report for the first time the occurrence of a mitochondria-targeted LEA gene (Afrlea3m) product in an animal species.

Disciplines

Biology

Publication Date

2009

Publisher Statement

This research was originally published in the Journal of Biological Chemistry. Menze, M. A., Boswell, L., Toner, M., and Hand, S. C. Occurrence of mitochondrial-targeted late embryogenesis abundant (LEA) gene in animals increases organelle resistance to water stress. Journal of Biological Chemistry. 2009. 284, 16:10714-10719. © the American Society for Biochemistry and Molecular Biology

Citation Information

Michael A Menze, Leaf Boswell, Mehmet Toner and Steven C. Hand. "Occurrence of Mitochondria-targeted Late Embryogenesis Abundant (LEA) Gene in Animals Increases Organelle Resistance to Water Stress" Journal of Biological Chemistry Vol. 284 Iss. 16 (2009)
Available at: http://works.bepress.com/michael_menze/11/